THE ORAL CIGARET

问题二、野良神第二季主题曲叫什么

片头曲《狂乱 Hey Kids!!》歌：口服香烟

相关问题三、谁知道野良神第二季片头曲，片尾曲的名字和含义，急求！谢谢！

片头曲《狂乱 Hey Kids!!》歌：口服香烟片尾曲念纳慎《仔敬》歌：茄链先谢了

你可能关注的问题四、请问下这个gif的出处

由日本方形Enix与角川(角川)两家公司首次合作推出的原创角色扮演游戏手游《Akashic Re：cords》近日发布了游戏过强；管理员视频100 . OP是日本人气摇滚乐团“口服香烟”所演唱的主题曲《抓住我》.本款实时对戟型RPG 《Akashic Re：cords》，除了由将会由日本人气作家”水野良”(代表作《罗德斯岛战记》 )负责本作剧情（由另一部由水野良专门为本作所撰写的原创小说登场)之外，还特别申请人气漫画家”中原”(代表作《FateGrand Order》 )负责主要人物设计，知名游戏作曲家”水田直志”(代表作《快打旋风Zero》、《光之四战士 FF外传》 )负责音乐在故事里，将会以后本书世界因此灭亡的”纸鱼”怪物入侵的巨大异界图书馆”怛魔书架”(恺魔的书架)为舞台，玩家们在游戏中将化身为一名接受召唤前保护图书馆的书籍，能自我创造”故事”的”创造者”,并召唤由自己创作的作品角色们登场，对抗各式各样的纸鱼强而本作最特别之处，就在于游戏中将会收录各式各样的召唤角色登场，而且这种召唤角色会有”小说（标签) /面向女性（少女) /科幻/恐怖（恐怖) /悬疑（推理) /幻想（幻想)”等六大类型不同的领域

推荐问题五、求一首日语歌，歌是女声，歌词一直是不要不要讨厌讨厌，她唱的时候还

“杉本艾玛”的”喜欢”我也找到了好久好久好久好久好久好久。评论第一句这位大姐，请尊重流产职业，不要开心笑容好吗歌词不行不行不行不行不行不行不行不行不行不行不行不行不行不行不行不行不行不行为研讨会随心所欲随心所欲地直抵清晨来临也毫无睡意地喜悦着那只鞋声，随心所欲地一直穿着那只脚踏舞声喜悦着

六、求英语作文。非常感谢！！！

风险因素许多癌症都与特定的环境和生活方式因素有关，这些因素使人容易患癌症。累积的数据表明，这些风险因素中的一些引发了癌症的发生，其他风险因素起到了促进作用，与有些危险因素会引发和促进疾病的发展。空气污染空气污染与癌症的发展有关，特别是肺癌。住在释放有毒化学物质的工厂附近的人患癌症的风险增加。许多有害物质，如砷、苯、碳氢化合物、聚氯乙烯和其他室内排放物也是如此

as vehicle exhaust ¡ª have been studied for their carcinogenic properties. Indoor air pollution, such as from cigarette smoke and radon, also poses an increased risk of cancer. In fact, indoor air pollution is considered to be more carcinogenic than outdoor air pollution. Tobacco Cigarette smoking increases the risk of lung cancer more than tenfold over that of nonsmokers by late middle age. Tobacco smoke contains nitrosamines and polycyclic hydrocarbons, two carcinogens that are known to cause mutations. The risk of lung cancer from cigarette smoking correlates directly with the ration of smoking and the number of cigarettes smoked per day. Tobacco smoke is also associated with laryngeal cancer and is considered a contributing factor in cancer of the bladder, pancreas, kidney, and cervix. Research also shows that a person who stops smoking decreases his or her risk of lung cancer. Although the risk associated with pipe and cigar smoking is similar to that of cigarette smoking, some evidence suggests that the effects are less severe. Smoke from cigars and pipes is more alkaline. This alkalinity decreases nicotine absorption in the lungs and also is more irritating to the lungs, so that the smoker doesn”t inhale as readily. Inhalation of “secondhand” smoke, or passive smoking, by nonsmokers also increases the risk of lung and other cancers. Plus use of smokeless tobacco, in which the oral tissue directly absorbs nicotine and other carcinogens, is linked to an increase in oral cancers that seldom occur in persons who don”t use the proct. Alcohol Alcohol consumption, especially in conjunction with cigarette smoking, is commonly associated with cirrhosis of the liver, a precursor to hepatocellular cancer. The risk of breast and colorectal cancers also increases with alcohol consumption. Possible mechanisms for breast cancer development include impaired removal of carcinogens by the liver, impaired immune response, and interference with cell membrane permeability of the breast tissue. Alcohol stimulates rectal cell proliferation in rats, an observation that may help explain the increased incidence of colorectal cancer in humans. Heavy use of alcohol and cigarette smoking synergistically increases the incidence of cancers of the mouth, larynx, pharynx, and esophagus. It”s likely that alcohol acts as a solvent for the carcinogenic substances found in smoke, enhancing their absorption. Sexual and reproctive behavior Sexual practices have been linked to specific types of cancer. The age of first sexual intercourse and the number of sexual partners are positively correlated with a woman”s risk of cervical cancer. Furthermore, a woman who has had only one sexual partner is at higher risk if that partner has had multiple partners. The suspected underlying mechanism here involves virus transmission, most likely human papilloma virus (HPV). HPV types 6 and 11 are associated with genital warts. HPV is the most common cause of abnormal Papanicolaou (Pap) smears, and cervical dysplasia is a direct precursor to squamous cell carcinoma of the cervix, both of which have been linked to HPV (especially types 16 and 31). Occupation Certain occupations, because of exposure to specific substances, increase the risk of cancer. Persons exposed to asbestos, such as insulation installers and miners, are at risk of a specific type of lung cancer. Asbestos also may act as a promoter for other carcinogens. Workers involved in the proction of dyes, rubber, paint, and beta-naphthylamine are at increased risk of bladder cancer. Ultraviolet radiation Exposure to ultraviolet radiation, or sunlight, causes genetic mutation in the P53 control gene. Sunlight also releases tumor necrosis factor alpha in exposed skin, possibly diminishing the immune response. Ultraviolet sunlight is a direct cause of basal and squamous cell cancers of the skin. The amount of exposure to ultraviolet radiation also correlates with the type of cancer that develops. For example, cumulative exposure to ultraviolet sunlight is associated with basal and squamous cell skin cancer, and severe episodes of burning and blistering at a young age are associated with melanoma. Ionizing radiation Ionizing radiation (such as X-rays) is associated with acute leukemia, thyroid, breast, lung, stomach, colon, and urinary tract cancers as well as multiple myeloma. Low doses can cause DNA mutations and chromosomal abnormalities, and large doses can inhibit cell division. This damage can directly affect carbohydrate, protein, lipid, and nucleic acids (macromolecules), or it can act on intracellular water to proce free radicals that damage the macromolecules. Ionizing radiation also can enhance the effects of genetic abnormalities. For example, it increases the risk of cancer in persons with a genetic abnormality that affects DNA repair mechanisms. Other compounding variables include the part and percentage of the body exposed, the person”s age, hormonal balance, prescribed drugs and preexisting or concurrent conditions. Hormones Hormones ¡ª specifically the sex steroid hormones estrogen, progesterone, and testosterone ¡ª have been implicated as promoters of breast, endometrial, ovarian, or prostate cancer. Estrogen, which stimulates the proliferation of breast and endometrial cells, is considered a promoter for breast and endometrial cancers. Prolonged exposure to estrogen, as in women with early menarche and late menopause, increases the risk of breast cancer. Likewise, long-term use of estrogen replacement without progesterone supplementation for menopausal symptoms increases a woman”s risk of endometrial cancer. Progesterone may play a protective role, counteracting estrogen”s stimulatory effects. The male sex hormones stimulate the growth of prostatic tissue. However, research fails to show an increased risk of prostatic cancer in men who take exogenous androgens. Diet Numerous aspects of diet are linked to an increase in cancer, including: obesity (in women only, possibly related to proction of estrogen by fatty tissue), which is linked to a suspected increased risk of endometrial cancer high consumption of dietary fat (e to an increase in free radical formation), which is linked to endometrial, breast, prostatic, ovarian, and rectal cancers high consumption of smoked foods and salted fish or meats and foods containing nitrites, which may be linked to gastric cancer naturally occurring carcinogens (such as hydrazines and aflatoxin) in foods, which are linked to liver cancer carcinogens proced by microorganisms stored in foods, which are linked to stomach cancer diet low in fiber (which slows transport through the gut), which is linked to colorectal cancer. ACS GUIDELINES: DIET, NUTRITION, AND CANCER PREVENTION ——————————————————————————– Because of the numerous aspects of diet and nutrition that may contribute to the development of cancer, the American Cancer Society (ACS) has developed a list of guidelines to rece cancer risk in persons ages 2 years and older. Choose most of the foods you eat from plant sources. ¨C Eat 5 or more servings of fruits and vegetables each day. ¨C Eat other foods from plant sources such as breads, cereals, grain procts, rice, pasta, or beans several times each day. Limit your intake of high-fat foods, particularly from animal sources. ¨C Choose foods low in fat. ¨C Limit consumption of meats, especially high-fat meats. Be physically active and achieve and maintain a healthy weight. ¨C Be at least moderately active for 30 minutes or more on most days of the week. ¨C Stay within your healthy weight range. Limit your consumption of alcoholic beverages, if you drink at all. The American Cancer Society (ACS) has developed specific nutritional guidelines for cancer prevention.